In the kidney, adenosine causes afferent glomerular arteriole constriction and its release position, by increasing the sodium load in the distal tubule, triggered as after furosemide administration. Therefore, adenosine may be a mediator of occurring deterioration of renal function with acute heart failure with acute heart failure be. In addition, it sodium chloride sodium chloride in the proximal tubule recovery. New equipmentantagonists are therefore currently under investigation with a view to preserving the renal function during the treatment of acute heart failure and diuretic and natriuretic diuretic and natriuretic effects of furosemide administration in patients with acute heart failure.

Heart disease. Failing kidney: Double Trouble?Concurrent renal failure and / or deterioration of renal function in patients with heart failure is a frequent finding and is associated with poor prognosis. Current heart failure treatment has beneficial effects on cardiac function, but not positive affect kidney function. Improve kidney function improve kidney function and / or kidney protection obtained with new drugs or devices is still uncertain.There are two major types of permanently dialysis access: an arteriovenous fistula, the artery and the vein a direct link and an arteriovenous transplant , which is connected, the artery and the vein a plastic tube. NIH , these compounds can take just between six and 12 months due stenosis and narrowing of the veins.

The Boston Globe: Leaders believe again a compromise bill be written by an House – Senate Conciliation Committee for, these the legislator will be more reluctant voting against it due of the historical character the bill .